An enhanced periodontal health status for adolescent orthodontic patients can be achieved through a specialized oral care mode.
Cone-beam computed tomography (CBCT) analysis of patients with temporomandibular dysfunction (TMD) and one-sided chewing patterns.
To form the experimental group, eighty patients with temporomandibular disorder (TMD) and unilateral chewing were chosen, and forty healthy volunteers were selected as the control group. Bilateral CBCT scans were performed on both groups to capture three-dimensional images, and subsequently, temporomandibular joint (TMJ) parameters were measured and compared in both groups. Data analysis was performed with SPSS 220 version of the software package.
A lack of substantial difference was found in bilateral TMJ parameters for the control group (P005). On the unilateral chewing side, the experimental group's condyles displayed a noticeably smaller inner and outer diameter compared to their counterparts on the non-unilateral chewing side, and a significantly elevated condyle horizontal angle and height (P<0.005). The experimental group exhibited significantly lower anteroposterior condyle diameter, inner and outer condyle diameters, horizontal and vertical condyle angles, intra-articular space, and post-articular space compared to the control group, whereas the pre-articular space was significantly higher (P<0.005). The condyle on the non-unilateral chewing side presented significantly diminished anteroposterior diameter and retro-articular space, compared to the controls. In contrast, the inner and outer diameters were substantially greater than those observed on the unilateral chewing side. Consistently, the condyle's height was markedly lower on the non-unilateral chewing side (P<0.005).
Due to unilateral chewing, individuals with TMD syndrome display unusual bilateral TMJ structures. These changes include a medial and posterior relocation of the condyle on the affected side, accompanied by a correlated expansion of the pre-articular space on the opposite side.
Individuals with TMD and unilateral mastication demonstrate structural changes in their bilateral temporomandibular joints. These changes manifest as medial and posterior condyle displacement on the affected side and a corresponding increase in pre-articular space on the unaffected side.
In order to establish a basis for evaluating the proficiency and performance appraisal methods of oral surgeons, a Delphi method will be used to create an appraisal system for the difficulty of oral surgery procedures.
To achieve expert selection, the Delphi method was applied across two rounds; simultaneously, a combination of critical value and synthetical index methods facilitated index selection; weighting for the index system was accomplished using a superiority chart.
A four-tiered, twenty-part index system was established to assess the difficulty of oral surgical procedures. The index system incorporated the concepts of index evaluation, index meaning, and index weight.
The oral surgery difficulty evaluation index system possesses unique characteristics when contrasted with conventional operation index systems.
A peculiar characteristic of the oral surgery difficulty evaluation index system distinguishes it from the traditional operation index system.
A clinical investigation of the effects of rapid maxillary expansion with cortical osteotomy, combined with orthodontic and orthognathic treatment, on skeletal Class III malocclusion.
A total of 84 skeletal Class malocclusion patients, admitted to Jining Dental Hospital between March 2018 and May 2020, were randomly assigned to an experimental group and a control group, each group containing 42 patients. While the control group underwent orthodontic-orthognathic treatment, the experimental group's treatment encompassed orthodontic-orthognathic treatment with rapid maxillary arch expansion achieved through cortical incision. Differences in the time needed to close gaps, align teeth, and the extent of maxillary first molar and central incisor movement along the sagittal axis were analyzed for both groups. Pre-treatment and four weeks post-treatment, measurements were taken to determine the vertical separations: from the upper central incisor's edge to the horizontal plane (U1I-HP), from the upper central incisor's apex to the coronal plane (U1I-CP), from the upper pressure groove edge to the coronal plane (Sd-CP), from the upper alveolar seat point to the horizontal plane (A-HP), from the upper lip's point to the coronal plane (Ls-CP), and from the inferior nasal point to the coronal plane (Sn-CP). These measurements allowed for the calculation of treatment-related changes. ISX-9 The treatment period witnessed a comparison of complications affecting the two groups. ISX-9 The statistical analysis of the data was performed using SPSS 200 software.
Alignment time, A-HP modification, Sn-CP adjustment, maxillary first molar migration distance, and maxillary central incisor displacement distance demonstrated no significant difference amongst the two groups (P005). The closing interval of the experimental group was markedly shorter than that of the control group, a finding that achieved statistical significance (P<0.005). The experimental group saw a considerably greater shift in U1I-HP, U1I-CP, Sd-CP, and Ls-CP when compared to the control group, which was statistically significant (P<0.05). The two groups experienced comparable complication rates during the treatment period, a finding substantiated by the non-significant p-value (P=0.005).
The integration of rapid maxillary expansion, cortical incision, and orthodontic-orthognathic procedures in patients with skeletal Class III malocclusions, can contribute to faster closing of the gap, superior treatment efficacy, and unaffected sagittal tooth positions.
Rapid maxillary expansion, achieved surgically through cortical incisions, combined with orthodontic and orthognathic treatment for skeletal Class III malocclusion, can effectively shorten the treatment timeframe while maintaining the teeth's sagittal alignment, yielding enhanced treatment outcomes.
The role of maxillary molars in influencing the thickening of the maxillary sinus mucosa was investigated using cone-beam computed tomography (CBCT).
The investigation incorporated 72 periodontitis patients and a subsequent CBCT analysis of 137 maxillary sinus instances. Evaluated parameters encompassed location, associated tooth, maximal mucosal thickness, alveolar bone loss, vertical intrabony pockets, and minimum residual bone height. Mucosal thickening was determined to be present in the maxillary sinus, with a thickness of 2 millimeters. ISX-9 The impact of various parameters on the dimensions of the maxillary sinus membrane was scrutinized. SPSS 250's functionalities, encompassing univariate analysis and binary logistic regression, were used to analyze the data.
Among 137 cases, mucosal thickening was found in 562%, increasing in prevalence as alveolar bone loss in the corresponding molar worsened, progressing from mild (211%) to moderate (561%) to severe (692%). The risk of maxillary sinus mucosal thickening escalated by 6-7 times for moderate bone loss (Odds Ratio = 713, 95% Confidence Interval = 137-3721), and a further significant increase for severe bone loss (Odds Ratio = 629, 95% Confidence Interval = 106-3737). The severity of intrabony pockets vertically aligned demonstrated a connection with mucosal thickness (no intrabony pockets 387%; type 634%; type 794%), which correlated to an increased risk of maxillary sinus mucosal thickening (type OR=372, 95%CI 101-1370; type OR=539, 95%CI 115-2530). The minimal residual bone height demonstrated a negative association with mucosal thickness (4 mm, odds ratio 9900, 95% confidence interval 1742-56279).
Maxillary molar alveolar bone loss, vertical intrabony pockets, and minimal residual bone height were found to be considerably linked to the thickening of the maxillary sinus mucosa.
Maxillary sinus mucosal thickening exhibited a significant association with indicators such as reduced alveolar bone level, vertical intrabony defects, and the lowest remaining bone height in maxillary molars.
We sought to explore the distribution of torque teno mini virus (TTMV) and Epstein-Barr virus (EBV) among patients with periodontitis.
Gingival tissue was gathered from a total of 80 patients who had periodontitis and 40 periodontally healthy volunteers. The presence of EBV and TTMV-222, as ascertained by nested PCR, was followed by real-time PCR quantification of the viral loads. Employing the SPSS 160 software package, a statistical analysis was conducted.
In the periodontitis cohort, detection rates and virus loads of EBV and TTMV-222 were found to be significantly elevated compared to the periodontal health group (P005). The detection rate for TTMV-222 was considerably higher in those with a positive EBV test compared to those without (P001). Significant evidence of a positive correlation between EBV and TTMV-222 was found in gingival tissue samples (P001).
Given the potential association between TTMV infection, co-infection with EBV, and periodontal disease, future studies should focus on deciphering the specific pathogenic mechanisms involved.
Potential links exist between TTMV infection and co-infection with EBV and TTMV and periodontal disease, but the pathogenic mechanisms of their mutual influence require further studies.
To ascertain the expression levels of semaphorin 4D (Sema4D) in bisphosphonate-related osteonecrosis of the jaw (BRONJ) and probe its possible involvement in BRONJ's etiology.
Intraperitoneal administration of zoledronic acid, assisted by tooth extraction, led to the development of a rat model with BRONJ-like features. The extraction of maxillary specimens for imaging and histological studies was performed, and subsequently, bone marrow mononuclear cells (BMMs) and bone marrow mesenchymal stem cells (BMSCs) were isolated from each group and subjected to in vitro co-culture. Trap staining and counting of monocytes commenced after osteoclast induction procedures were completed. Following osteoclast orientation, RAW2647 cells cultivated in a bisphosphonates (BPs) environment exhibited demonstrable Sema4D expression. In a parallel manner, MC3T3-E1 cells and bone marrow stromal cells underwent osteogenic induction in vitro, and the expression profile of osteogenic and osteoclastic genes, such as ALP, Runx2, and RANKL, was quantitatively determined under the influence of bisphosphonate treatments, Sema4D exposure, and the addition of a Sema4D neutralizing antibody.